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    • Alpha Actinin antibodies
    • Anti- Virus ANTIBODIES
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    • Interleukin & Growth Factor Receptor Data Pack
    • ORDERING INFORMATION
    • paramethasone thru polacrilex resin - Research Diagnostics
    • POLY-HRP STREPTAVIDIN
    • Polyclonal Antibody TO 5'-BROMO-2'DEOXYURIDINE
    • Press Release from Research Diagnsotisc Inc
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  • Blog
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    • Angiopoietin ANTISERA-Human IgG Subclasses Book Section 5.3 & 5.4
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  • HBsAg Antibody | Gentaur
  • HBsAg Antibody
  • HBsAg Antibody

    HBsAg Antibody | Gentaur

    Gentaur

    MSRP:
    Now: €340.00
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    SKU:
    13-ORB23041-GEN
    Availability:
    IN STOCK
    Size:
    100 µg

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    Product Description

    HBsAg Antibody

    Quality Level

    100

    biological source

    mouse

    conjugate

    unconjugated

    antibody form

    purified immunoglobulin

    antibody product type

    primary antibodies

    clone

    HB5, monoclonal

    form

    buffered aqueous solution

    species reactivity

    hepatitis B virus

    concentration

    1 mg/mL

    technique(s)

    ELISA: suitable
    immunocytochemistry: suitable

    Hepatitis B surface antigen (HBSAg) and antibodies to both the surface and core antigens of the hepatitis B virus (anti-HBS and anti-HBC) have been studied in 64 consecutive cases of fulminant hepatitis. HBSAg was detected by counterelectrophoresis in 23 (35-9%) but by radioimmunoassay in 38 (59-3%). Anti-HBS was detected by passive haemagglutination in 26 (40-6%), coexisting HBSAg and anti-HBS were found in 16 cases (25%).

    Using an indirect immunofluorescence technique, anti-HBC was found in all of the cases in whom either HBSAg or anti-HBS was present. The highest survival rate was observed in patients with no evidence of HBV infection (31-3%) and was lowest in those who had both HBSAg and anti-HBS detected simultaneously (6-2%).

    The prognosis of those who exhibited anti-HBS only was no better than those with HBSAg alone. In a further case, transient interruption of the asymptomatic chronic HBSAg carrier state with seroconversion to anti-HBS was associated with the development of a fulminant hepatitis syndrome.

    The results suggest that an unusually strong and rapid immune clearance of HBSAg may be involved in the pathogenesis of fulminant hepatitis.

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